AMPLIFICATION OF THE PRO-FIBROTIC JAK2-STAT3 SIGNALING AXIS BY TGFB-INDUCED EPIGENETIC SILENCING OF SOCS3

Dees C, Poetter S, Zhang Y, Bergmann C, Zhou X, Luber M, Karouzakis E, Ramming A, Distler O, Schett G, Distler J (2020)

Publication Type: Conference contribution

Publication year: 2020

Journal

Annals of the Rheumatic Diseases BMJ Publishing Group

Publisher: BMJ PUBLISHING GROUP

City/Town: LONDON

Pages Range: 1084-1084

Conference Proceedings Title: ANNALS OF THE RHEUMATIC DISEASES

Event location: , ELECTR NETWORK

DOI: 10.1136/annrheumdis-2020-eular.4489

Authors with CRIS profile

Clara Dees Department of Medicine 3 – Rheumatology and Immunology Yun Zhang Department of Medicine 3 – Rheumatology and Immunology Andreas Ramming Medizinische Fakultät Georg Schett Lehrstuhl für Innere Medizin III Jörg Distler Professur für Molekulare Mechanismen der Organfibrose

Involved external institutions

Universitätsspital Zürich (USZ) CH Switzerland (CH)

How to cite

APA:

Dees, C., Poetter, S., Zhang, Y., Bergmann, C., Zhou, X., Luber, M.,... Distler, J. (2020). AMPLIFICATION OF THE PRO-FIBROTIC JAK2-STAT3 SIGNALING AXIS BY TGFB-INDUCED EPIGENETIC SILENCING OF SOCS3. In ANNALS OF THE RHEUMATIC DISEASES (pp. 1084-1084). , ELECTR NETWORK: LONDON: BMJ PUBLISHING GROUP.

MLA:

Dees, Clara, et al. "AMPLIFICATION OF THE PRO-FIBROTIC JAK2-STAT3 SIGNALING AXIS BY TGFB-INDUCED EPIGENETIC SILENCING OF SOCS3." Proceedings of the Annual European Congress of Rheumatology (EULAR), , ELECTR NETWORK LONDON: BMJ PUBLISHING GROUP, 2020. 1084-1084.

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