ACTN4 p.Ile150Met Causes FSGS With Validation in Primary Fibroblasts and Immortalized Podocytes

Grosch M, Haak J, Kolb C, Plogmann I, Guaragna MS, Wiesener A, Pasutto F, Wopperer F, Pollinger L, Engel FB, Wiesener M, Schiffer M, Jobst-Schwan T (2026)

Publication Type: Journal article

Publication year: 2026

Journal

Kidney International Reports Elsevier

Book Volume: 11

Article Number: 103706

Journal Issue: 2

DOI: 10.1016/j.ekir.2025.11.030

Authors with CRIS profile

Melanie Grosch Department of Medicine 4 – Nephrology and Hypertension Jan Haak Department of Medicine 4 – Nephrology and Hypertension Francesca Pasutto Institute of Human Genetics Florian Wopperer Department of Medicine 4 – Nephrology and Hypertension Lena Pollinger Department of Medicine 4 – Nephrology and Hypertension Michael Wiesener Professur für Innere Medizin (Nephrologie) Mario Schiffer Lehrstuhl für Innere Medizin IV Tilman Jobst-Schwan Department of Medicine 4 – Nephrology and Hypertension

Involved external institutions

Universidade Estadual de Campinas (UNICAMP) / University of Campinas BR Brazil (BR) Klinikum rechts der Isar DE Germany (DE)

How to cite

APA:

Grosch, M., Haak, J., Kolb, C., Plogmann, I., Guaragna, M.S., Wiesener, A.,... Jobst-Schwan, T. (2026). ACTN4 p.Ile150Met Causes FSGS With Validation in Primary Fibroblasts and Immortalized Podocytes. Kidney International Reports, 11(2). https://doi.org/10.1016/j.ekir.2025.11.030

MLA:

Grosch, Melanie, et al. "ACTN4 p.Ile150Met Causes FSGS With Validation in Primary Fibroblasts and Immortalized Podocytes." Kidney International Reports 11.2 (2026).

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