Silke Härteis

Lehrstuhl für Physiologie (Vegetative Physiologie)

Proteolytic activation of the epithelial sodium channel (ENaC) by factor VII activating protease (FSAP) and its relevance for sodium retention in nephrotic mice (2021) Artunc F, Bohnert BN, Schneider JC, Staudner T, Sure F, Ilyaskin A, Worn M, et al. Journal article Bile acids inhibit human purinergic receptor P2X4 in a heterologous expression system (2019) Ilyaskin A, Sure F, Nesterov V, Härteis S, Korbmacher C Journal article The degenerin region of the human bile acid-sensitive ion channel (BASIC) is involved in channel inhibition by calcium and activation by bile acids. (2018) Ilyaskin A, Kirsch S, Böckmann R, Sticht H, Korbmacher C, Härteis S, Diakov A Journal article, Original article Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice. (2018) Härteis S, Schork A, Doerffel T, Bohnert BN, Nacken R, Woern M, Xiao M, et al. Journal article Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome (2018) Bohnert BN, Menacher M, Janessa A, Woern M, Schork A, Daiminger S, Kalbacher H, et al. Journal article Erratum to: Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase. (2017) Sostegni S, Diakov A, Mcintyre P, Bunnett N, Korbmacher C, Härteis S Journal article, Erratum Bile acids potentiate proton-activated currents in Xenopus laevis oocytes expressing human acid-sensing ion channel (ASIC1a). (2017) Ilyaskin A, Diakov A, Korbmacher C, Härteis S Journal article Activation of the Human Epithelial Sodium Channel (ENaC) by Bile Acids Involves the Degenerin Site (2016) Ilyaskin A, Diakov A, Korbmacher C, Härteis S Journal article Association of Plasminuria with Overhydration in Patients with CKD (2016) Schork A, Woern M, Kalbacher H, Voelter W, Nacken R, Bertog M, Härteis S, et al. Journal article Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain (2015) Zhao P, Lieu T, Barlow N, Sostegni S, Härteis S, Korbmacher C, Liedtke W, et al. Journal article