Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation

Tegtmeyer N, Backert S (2017)

Publication Status: Published

Publication Type: Journal article, Letter

Publication year: 2017

Journal

Disease Models & Mechanisms Company of Biologists: OAJ / Company of Biologists

Publisher: Company of Biologists Ltd

Book Volume: 10

Pages Range: 1539-1540

Journal Issue: 12

DOI: 10.1242/dmm.032813

Authors with CRIS profile

Nicole Tegtmeyer-Backert Lehrstuhl für Mikrobiologie Steffen Backert Lehrstuhl für Mikrobiologie

How to cite

APA:

Tegtmeyer, N., & Backert, S. (2017). Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation. Disease Models & Mechanisms, 10(12), 1539-1540. https://dx.doi.org/10.1242/dmm.032813

MLA:

Tegtmeyer, Nicole, and Steffen Backert. "Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation." Disease Models & Mechanisms 10.12 (2017): 1539-1540.

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