Differential Levels and Phosphorylation of Type 1 Inositol 1,4,5-Trisphosphate Receptor in Four Different Murine Models of Huntington Disease
Post JI, Leergaard TB, Ratz V, Walaas SI, von Hörsten S, Nissen-Meyer LSH (2019)
Publication Type: Journal article
Publication year: 2019
Journal
Book Volume: 8
Pages Range: 271-289
Journal Issue: 3
DOI: 10.3233/JHD-180301
Abstract
Background: The intracellular ion channel type 1 inositol 1,4,5-trisphosphate receptor (IP(3)R1) releases Ca-2(+) from the endoplasmic reticulum upon stimulation with IP3. Perturbation of IP(3)R1 has been implicated in the development of several neurodegenerative disorders, including Huntington disease (HD).
Authors with CRIS profile
Veronika Ratz-Wirsching
Department of Experimental Therapy
Stephan von Hörsten
Professur für Experimentelle Biomedizin
Involved external institutions
Norway (NO)How to cite
APA:
Post, J.I., Leergaard, T.B., Ratz, V., Walaas, S.I., von Hörsten, S., & Nissen-Meyer, L.S.H. (2019). Differential Levels and Phosphorylation of Type 1 Inositol 1,4,5-Trisphosphate Receptor in Four Different Murine Models of Huntington Disease. Journal of Huntington's Disease, 8(3), 271-289. https://dx.doi.org/10.3233/JHD-180301
MLA:
Post, Joakim Iver, et al. "Differential Levels and Phosphorylation of Type 1 Inositol 1,4,5-Trisphosphate Receptor in Four Different Murine Models of Huntington Disease." Journal of Huntington's Disease 8.3 (2019): 271-289.
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