TGF-beta-Induced Epigenetic Silencing of SOCS3 Facilitates STAT3 Signaling to Promote Fibroblast Activation and Tissue Fibrosis in Systemic Sclerosis

Dees C, Zhang Y, Pötter S, Bergmann C, Distler O, Schett G, Distler J, Ramming A (2017)

Publication Type: Conference contribution

Publication year: 2017

Journal

Arthritis and Rheumatology Wiley

Book Volume: 69

Authors with CRIS profile

Clara Dees Department of Medicine 3 – Rheumatology and Immunology Yun Zhang Department of Medicine 3 – Rheumatology and Immunology Sebastian Pötter Department of Medicine 3 – Rheumatology and Immunology Georg Schett Lehrstuhl für Innere Medizin III Jörg Distler Professur für Molekulare Mechanismen der Organfibrose Andreas Ramming Department of Medicine 3 – Rheumatology and Immunology

Involved external institutions

Universitätsspital Zürich (USZ) CH Switzerland (CH)

How to cite

APA:

Dees, C., Zhang, Y., Pötter, S., Bergmann, C., Distler, O., Schett, G.,... Ramming, A. (2017). TGF-beta-Induced Epigenetic Silencing of SOCS3 Facilitates STAT3 Signaling to Promote Fibroblast Activation and Tissue Fibrosis in Systemic Sclerosis.

MLA:

Dees, Clara, et al. "TGF-beta-Induced Epigenetic Silencing of SOCS3 Facilitates STAT3 Signaling to Promote Fibroblast Activation and Tissue Fibrosis in Systemic Sclerosis." 2017.

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